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Tuesday, December 28, 2010

More On Aricept, Ginkgo Biloba and My bvFTD

Everything here is my account of what happened to me, or my interpretation of stuff. Every case of FTD (Frontotemporal Dementia - Pick's) is different. Keep in mind as you read this that the person who wrote this has dementia - bvFTD. That would be ME.
Medical Disclaimer 

Making big news at the end of 2008 was a six-year research study reported in the November 19, 2008 issue of The Journal of the American Medical Association (JAMA). It found that Ginkgo Biloba was not any more effective for the prevention of Alzheimer’s disease or other dementia than a placebo.

The 3069 subjects, all over age 75, took EGb 761 twice a day (total 240 mg) or a placebo. Let me interject here that EGb761 is one of the supposed active compounds in Ginkgo Biloba, but not the only one. Anyway -  Study volunteers had normal awareness or very mild impairment at the start of the trial. Many news stories have seemed to generalize this research trial, and wrongly discount Ginkgo Biloba for any beneficial use.

Ginkgo Biloba has been all over the news again lately, and all of the stories seem to be based on the same study from 2008 because it was just published in November of 2010 by the The Journal of the American Medical Association (JAMA). The headline usually reads something like,"Ginkgo is not effective in treating Dementia". Well -  as the renowned Dr. Joyce Brothers was always so fond of saying, "One study shows... ". 

It is all the same study. Most of the news stories mis-interpreted the results of the study. When I researched the actual study myself I found that it was performed on an extremely aged sample of people (age 75+!) who were healthy (as in No Dementia!), and it showed that Ginkgo did not significantly prevent dementia over the six year period when they were followed. The dose of Ginkgo administered was the minimum of 120 mg, while the recommended dose for anyone with dementia is 240 mg to as much as 600 mg per day. The study also found no adverse effects when taking Ginkgo Biloba which is consistent with most other studies. Over all it was a very well-done study with a huge sample size, and the conclusions are quite probably correct that Ginkgo Biloba will not prevent dementia. I must mention that there was another study published in a less prestigious journal at the exact same time as this one which came to the exact opposite conclusion. Well... the study was great, but the way it was frequently reported in the media is crap.

My reaction to all that is... So What! I already have dementia, so I don't care if Ginkgo prevents it. Too late for that!

My question is:
Will Ginkgo Biloba help with the symptoms I have now, or slow the progression of my dementia?

Ahhhh... now that seems to be a totally different story. Out of over 125 research studies, many of which are very well done, the overwhelming conclusion is that Ginkgo Biloba has a significant beneficial effect on dementia symptoms. No, I did not review all 125+ studies myself, but I did find several review articles that did that for me which is just as good.

Over a hundred studies all say that Ginkgo Biloba has beneficial effects if you have dementia. Duh! Go do the research for yourself, and then - go Ginkgo!

This is a no-brainer. Since I have dementia, and I do not suffer from seizures, I am going to take Ginkgo Biloba. The fact that it is very inexpensive compared to other medications and supplements is just an added bonus.

So, since I also take Aricept, I was concerned about an interaction. It turns out that there might be one: Ginkgo may act in synergy with Aricept. That means that it could make any side effects of Aricept worse, so is not recommended. It also means that it should enhance any beneficial effects. Since my nasty side effects from Aricept have gone away, I see a clear advantage to taking both if I can tolerate them.

A research study that appeared in the September 2006 issue of the European Journal of Neurology compared EGb 761 to the common drug used to treat mild to moderate Alzheimer’s disease –donepezil (trade name Aricept). The six-month study began with 76 people, ages 50 to 80, who were divided into three groups, those who took EGb 761 (160 mg per day), donepezil (5 mg per day) or a placebo.

The results for the 60 individuals who finished the study showed a significant improvement in attention and memory for the Ginkgo and donepezil groups over the placebo group. In fact, the researchers noted that Ginkgo and donepezil produced the same improved-memory results. Furthermore, the Ginkgo group was side-effect-free compared to the donepezil group that had reported diarrhea or nausea symptoms, and Ginkgo, of course, was considerably less expensive. Note that in this study the 2 were not combined, they were compared.

So, even though the above study may indicate that Ginkgo is just as effective as Aricept, and other studies indicate the two may be synergistic, I would not expect the final outcome to be an effect twice as beneficial as the two taken individually. It just never works that way in real life. I would, however, fully expect that when taking the two together the beneficial effects would be better than either one taken alone.

So, the bottom line is that I am going to try to combine the traditional drug Aricept with the alternative medicine Ginkgo Biloba, and hopefully increase the benefits of both.

So far, after several weeks, that seems to be the case. Though I recently am trying an increased dose of Aricept (23 mg) I have not had any severe side effects. A little nausea, and occasionally a feeling of "jitteryness", but nothing to outweigh the benefits. I am currently taking 120 mg of Ginkgo Biloba Plus twice daily (morning and night) for a total of 240 mg of Ginkgo, and 23 mg of Aricept each morning.

Since I have been taking the combination I feel like I can think again!

No, it has not alleviated all of my symptoms. I still have difficulty with initiation, working memory, and motivation. As far as cognition goes, I have seen a huge improvement. Thinking isn't as difficult as it was a year ago. I no longer need a daily to-do list. I can do some simple mental arithmetic. I am not pausing to search for words as often. I can play video games competitively again! These are huge positive changes compared to a year ago. Again, I am not symptom-free, but I am improved from a year ago.

Of course, this evaluation is subjective. I cannot test or measure any improvement. Others who know me also report seeing a huge improvement. That is the best I have to go on.

The next Alternative Medications to add to my cocktail are... Melatonin and mushrooms. More on that later.

Comments are welcome.

Wednesday, December 22, 2010

Aricept - Aricept and My bvFTD - Update 1

Aricept is not approved for bvFTD, but it seems to be helping me... A whole bunch!

Everything here is my account of what happened to me, or my interpretation of stuff. Every case of FTD is different. Keep in mind as you read this that the person who wrote this has dementia. That would be ME.
Medical Disclaimer.

This is a short update to the first post about my taking Aricept for bvFTD. I started taking Aricept 5 mg on September 13, 2010. I increased the dosage to 10 mg after about 6 weeks, and now on December 13, 2010 I increased the dosage to 23 mg. That was 7 days ago.

Initially when I first started taking it, I had some severe difficulties with the side effects of Aricept. Though I did not seem to have any adverse behavioral issues, I did have some gastrointestinal problems the most bothersome of which was heartburn. The insomnia was not too bad, but the itching was horrible, and almost caused me to discontinue taking the drug. I cannot express in words how severe the itching was - think poison ivy on top of mosquito bites... and add a few chiggers! Then multiply by two!

Finally! All of the nasty side effects have gone away. They lasted about 4 - 6 weeks, and were considerably more manageable near the end of that period. There may still be a little nausea and insomnia now and then, but that may have nothing to do with the Aricept because of some other medications I occasionally take which also can cause both nausea and insomnia. In any case it does not happen often, doesn't last more than a few minutes,  and is not severe.

With the approval of my neurologist I have increased the dosage again over the past week to see if I can tolerate the higher 23 mg dosage without having a return of the adverse side effects. So far it has only been a few days, so it is too early to say anything conclusive about that. If it is like when I first started taking Aricept 3 months ago then I would expect to have any side effects start to show up over the next 2 - 3 weeks. I am hopeful they will be short-lived if at all.

Though Aricept is not approved for treatment of bvFTD it seems to be helping me a lot with no adverse behavioral effects that are apparent to me.

Some beneficial changes in me that I and others have noted in the past 12 weeks since taking Aricept include: being able to do mental math again, not using a daily to-do list, quicker thinking and speech. Generally more like my "old self".

I don't really have too many ways to objectively evaluate any benefits. I can say that without a doubt I feel like my thinking is sharper, and my cognition has improved. Subjectively I think I have seen a huge improvement regarding my Dysexecutive Syndrome, and maybe a slight improvement in attention. My only objective test is that I can now add a couple numbers in my head, and do other mental math. As an example: I was at a store a couple days ago, and as I was being checked out I was adding the stuff up in my head as it was being tallied into the cash register. When the total was given, I was confident enough in my math capability to question it because it was about $22 higher than what I had added it up to be. I was right! I had been double charged for a bag of a hundred wine corks!

I could not have done that a year ago! So, in my opinion, Aricept is helping. I am also taking a couple of herbal supplements as well, specifically Ginkgo and Melatonin. I strongly believe that the combination of the Ginkgo and the Aricept is having a huge impact. The two have been shown to interact in numerous studies, and as soon as I started taking the Ginkgo Biloba I noticed an increase in the effects of the Aricept. Maybe it is a coincidence... if you believe in that sort of thing. I will post more on this soon.

All of that is just a bonus. I am not taking Aricept to improve my FTD symptoms. My purpose in taking it is to slow or stop the progression of my bvFTD. If I can gain even a couple years any nasty side-effects (except the itching!) would be well worth it. Unfortunately there is no way I can think of to objectively measure if it is working to slow the progression or not. Only time will tell.

So, for now I am going to continue taking the Aricept, and also continue to explore the options of Alternative Medicine.

Comments are welcome

Nicotine and Dementia - Cigar anyone?

"A woman is an occasional pleasure but a cigar
is always a smoke.” - Groucho Marx 

I smoke a cigar now and then. Occasionally, some ill-informed non-smoking do-gooder tries to explain, usually at great length, just how bad smoking is for my health. I wait patiently as I enjoy the unparalleled pleasures of my cigar, and when they eventually finish, sure that they have convinced me, I reply, "I will die from complications of dementia long before any possible result of cigar smoking. In addition, nicotine in the form of my cigar serves to alleviate my symptoms, and is in effect a treatment for my disease. Want another drink?" 

...and for those really annoying people who need proof:

Nicotine clues for dementia treatment

14. July 2008 15:35

A team of London scientists have found clues for the potentially therapeutic benefits of nicotine on learning, memory and attention while minimising the risk of addiction. The research announced in Geneva Monday will assist the search for new drugs for dementia.

"Nicotine, like many other drugs, has multiple effects some of which are harmful whereas others may be beneficial," said Professor Ian Stolerman from the Institute of Psychiatry, King's College London. Previous research has revealed these cognitive effects in humans and in laboratory animals. "They are small effects and," he warned, "for healthy people they do not outweigh the harmful effects." The pharmaceutical industry has striven to discover nicotine-like substances for conditions such as Alzheimer's disease. Nicotine itself is difficult to administer by conventional means. The differences between doses that produce cognitive and toxic effects are small and, most significantly, there is also high risk of addiction. The balance, however, between costs and benefits is much more favourable for people with serious illnesses such as dementia. Speaking at Europe's biggest neuroscience conference, Professor Stolerman explained that newer substances are based upon the chemical structure of the nicotine molecule. Research in rats has shown a nicotine-induced improvement in sustained attention to visual stimuli. The King's College team studied the underlying mechanisms that produced this change and have helped to identify the roles of nicotinic receptors - the proteins on cells that respond to nicotine - as well as the involvement of several chemicals in the brain, including dopamine, noradrenaline, glutamate and serotonin. "We found several similarities and only small differences between the cognitive mechanisms and those involved in the addictive effects of nicotine," said Professor Stolerman. "The cognitive 'boost' that many smokers experience from nicotine probably contributes to the reason people smoke cigarettes, so it may not be possible to totally prevent addiction. Nevertheless, the potential for abuse of a medicine based on a pure nicotine-like substance is likely to be very small." The new knowledge about mechanisms of nicotine action may speed the discovery of agents that are more effective as cognitive enhancers than nicotine itself, with longer-lasting effects. "This is a promising stage in the years of research that have endeavoured to separate the beneficial from the harmful effects of nicotine," Professor Stolerman concluded. 

But, more importantly, a beautiful woman recently remarked over a glass of fine apple wine how she loved the smell of my cigar, and in spite of anything Groucho may have thought, that alone would be reason enough to smoke one now and then. 

So my advice is,  if you don't smoke, don't start, but if you have dementia, who gives a shit? 

 Comments are welcome.

Friday, December 17, 2010

My Brain With bvFTD - All About Memory

A Technical Look At Short-Term Memory

Since one of my main difficulties is with Working Memory I wanted to know more about it. There has been a large amount of research on memory since I took my last Psychology class. About all I can remember of brain anatomy is... Brain Stem! Brain Stem!

The following is copied directly with permission from the web site, The Brain From Top To Bottom. It is a great site, so expect to see more in the future.


In the course of a day, there are many times when you need to keep some piece of information in your head for just a few seconds. Maybe it is a number that you are “carrying over” to do a subtraction, or a persuasive argument that you are going to make as soon as the other person finishes talking. Either way, you are using your short-term memory.

In fact, those are two very good examples of why you usually hold information in your short-term memory: to accomplish something that you have planned to do. Perhaps the most extreme example of short-term memory is a chess master who can explore several possible solutions mentally before choosing the one that will lead to checkmate.

This ability to hold on to a piece of information temporarily in order to complete a task is specifically human. It causes certain regions of the brain to become very active, in particular the pre-frontal lobe.

 This region, at the very front of the brain, is highly developed in humans. It is the reason that we have such high, upright foreheads, compared with the receding foreheads of our cousins the apes. Hence it is no surprise that the part of the brain that seems most active during one of the most human of activities is located precisely in this prefrontal region that is well developed only in human beings.

Human memory is a complex phenomenon, however, and of course involves other regions of the brain as well.

(Brain Stem! Brain Stem!)


Baddeley’s model of working memory has proven especially fruitful for research on the brain areas involved. This model posits a central processor that coordinates the activity of two sub-systems. Many brain-imaging studies show high activity in the frontal lobe when this central processor is working.

Source: NIMH Laboratory of Brain and Cognition. Published in Nature, Vol 386, April 10, 1997, p. 610
For example, the image shown here was produced by functional magnetic resonance, a technique based on the increased blood flow to the most active areas of the brain. In this image, taken while the subject was holding an image of a face in his memory, the yellow area in the prefrontal cortex is very active.

But Baddeley’s model also postulates the existence of a phonological (acoustic and linguistic) memory and a visual/spatial memory (containing mental images). Brain imaging studies have also revealed distinct neuroanatomical bases for both of these forms of memory.

The phonological loop activates certain areas in the left hemisphere that are associated with the production of language, such as Wernicke’s area and Broca’s area. Visual/spatial memory seems to be associated with a region of the occipital cortex generally associated with visual processing.

Meanwhile, certain sub-regions of the prefrontal cortex are activated only if the memorization exercise is somewhat difficult for the subject, thus confirming the coordinating role of the central processor.
Things get even more complicated when you consider the chronological sequence of memorization: the various steps involved in storing and retrieving a piece of information.


The hippocampus, the cortical structures surrounding it, and the neural pathways that connect them to the cortex as a whole are all heavily involved in declarative memory–the memory of facts and events.

For example, after you’ve had a fine dinner with some friends, your memories of their faces, the taste of the wine, and the music that was playing are distributed in the various visual, olfactory, and auditory areas of the brain, but they are all connected together by the hippocampus to form an "episode", rather than remaining a collection of separate memories.

The hippocampus thus plays a fundamental role in episodic memory, the kind that will let you remember this especially pleasant dinner party years later. In fact, it seems to be the hippocampus that enables you to “play the scene back”, by reactivating this particular activity pattern in the various regions of the cortex. This phenomenon would be very important during dreams, and would explain the incorporation of events from the last few days into them.

But after a while, these various cortical regions activated during an event would become so strongly linked with one another that they would no longer need the hippocampus to act as their link. 

Thanks to this linkage, the memory of a piece of music that was playing that night could be enough to bring back the entire scene of the dinner party. Each of these elements could act as an index entry that lets you retrieve all the others to your consciousness.

Thus, information that has been encoded in long-term memory for a lengthy period of time no longer requires the intervention of the hippocampus. This is the case in particular for general knowledge in semantic memory, which instead activates the frontal and temporal cortexes. The activity in the temporal lobe would correspond to the activation of the fact in question, while the activity in the frontal cortex would correspond to its reaching consciousness.

Unlike our memory of facts and events, however, our spatial memory appears to be confined to the hippocampus. And more specifically to the right hippocampus. This structure seems to be able to create a mental map of space, thanks to certain cells called place cells.

Some very intense personal memories that bring what is sometimes called emotional memory into play appear to involve another structure of the limbic system besides the hippocampus. This structure is the amygdala, which is already known to manage our reactions to fear. Many other structures in the limbic system also help to encode our long-term memories.

Lastly, procedural memory, such as knowing how to ride a bike, does not appear to involve the hippocampus at all. Instead, procedural memory appears to be associated with modifications in the cerebellum, the basal ganglia, and the motor cortex, all of which are involved in motor control. As evidence to this effect, procedural memory is not affected by amnesia caused by lesions to the hippocampus, but is affected by damage to the cerebellum and by neurodegenerative diseases that alter the basal ganglia, such as Huntington’s disease.

But wait! There's more... BRAIN STEM! BRAIN STEM!


Sensory memory is the memory that results from our perceptions automatically and generally disappears in less than a second. It includes two sub-systems: iconic memory of visual perceptions and echoic memory of auditory perceptions.  
Short-term memory depends on the attention paid to the elements of sensory memory. Short-term memory lets you retain a piece of information for less than a minute and retrieve it during this time. One typical example of its use is the task of repeating a list of items that has just been read to you, in their original order. In general, you can retain 5 to 9 items (or, as it is often put, 7±2 items) in short-term memory.

Working memory is a more recent extension of the concept of short-term memory. As techniques for studying memory have become more refined, it has become increasingly apparent that the original conception of short-term memory as a mere temporary receptacle for long-term memory is too simplistic. In fact, it is becoming increasingly clear that there is no strict line of demarcation between memories and thoughts. In order to test some hypotheses that may provide a better understanding of this complex phenomenon, the concept of working memory has therefore been advanced.

Working memory is used to perform cognitive processes on the items that are temporarily stored in it. It would therefore be heavily involved in processes that require reasoning, such as reading, or writing, or performing computations. One typical example of the use of working memory is the task of repeating a list of items that has just been read to you, but in the reverse of their original order.

Another good example is the task of simultaneous interpretation, where the interpreter must store information in one language while orally translating it into another.

Working memory appears to be composed of several independent systems, which would imply that we are not aware of all the information that is stored in it at any given time. For example, when you drive a car, you are performing several complex tasks simultaneously. It is unlikely that all of the various types of information involved are being handled by a single short-term memory system.

The content of  this post - All About Memory - is under copyleft.

The concept of "copyleft" is a method of providing free access to the results of original work and of encouraging people to reproduce and even modify this work on an equally free basis.

Copyleft is thus diametrically opposed to the traditional concept of copyright, which nowadays people seem to be trying to use to cover absolutely everything, from genes to intellectual property. In the libertarian spirit, the concept of copyleft promotes freedom of expression and staunchly opposes the idea that knowledge can be the private property of a small elite. So, feel free to copy or link to the contents of this post for noncommercial use.

BRAIN STEM! BRAIN STEM! ...make it stop....

Comments are welcome.

Tuesday, December 14, 2010

My Brain With bvFTD - Parts Of The Brain


I wanted to review the general parts of the brain before going into some detail on working memory, and here is the absolute best.


Comments are welcome.

Tuesday, December 7, 2010

FTD: It Could Be worse...

...it could be raining!

“It's better to be an optimist who is sometimes wrong than a pessimist who is always right”
“In the long run the pessimist may be proved right, but the optimist has a better time on the trip.”  - Daniel L. Reardon 

So... everyplace I look I see the same things:

There is no cure for Frontotemporal Dementia.

There is no treatment for Frontotemporal Dementia.

There are no approved drugs for Frontotemporal Dementia.

Frontotemporal Dementia is a progressive disease.  Yadda... Yadda... Yadda... and then you die!

No matter what you call it, Pick's Disease, FTD, bvFTD, FTLD, or  Frontotemporal Dementia it doesn't matter.  At this time nobody knows much about it. Current research is just beginning to unravel the chemical pathways in the brain that lead to the atrophy of the frontal and temporal lobes. Nobody yet knows exactly what causes it, or why it happens. Doctors currently cannot even agree on precisely how to describe it, or to even diagnose it. To oversimplify it, it is known that there is some breakdown of some kind for some reason of the pathway involving Tau Proteins in the brain causing aggregations and atrophy. Not much to go on, but more is being learned about it every day.

So what does all this pessimistic crap mean to me with FTD?

1. There is no cure. OK, all that means to me is that the FDA and the AMA etc. have not approved a cure. The research is still inconclusive, and barely in its infancy.  I am sure there actually is a cure out there somewhere just waiting to be recognized. This does not by any means say that FTD is incurable, just that at this time there is no standardized approved treatment. Which leads to...

2.There is no treatment. Actually there are many treatments, but none of them are recognized to be effective at this time by the health care industry. Almost everything that is looked at in the research seems to have some beneficial effect. Seriously! If they look at exercise, it seems to help. If they look at Ginkgo Biloba, it seems to help. If they look at the drugs approved for Alzheimer's, they seem to help. If they look at other herbal remedies, they seem to help. When they look at pain killers, massages,  aromatherapy, prayer, Diabetes Drugs (Metformin), wine, sex, vitamins, hugging your pet - whatever - it seems to help. The problem is that when someone else looks at the exact same thing it does not help. Every case of FTD is different. So what this is telling me is that every case of FTD probably needs to be treated differently. Duh! This is never going to work in our current health care system where Doctors treatments are standardized, and usually dictated by the insurance companies and other medical institutions. Maybe some day there will be an approved treatment, but until then I am on my own to figure out what if anything works for me and my particular flavor of Lime Jell-O. Until there is a treatment for what causes FTD the best I can do is try to figure out if anything helps to manage my symptoms. If I am lucky something that helps with the symptoms might also help with the cause. So what if the odds are against it, and the probability is low? I have already found some things that seem to help, and some that do not. I already have started to develop an approved treatment for me - approved by me. So, do not tell me that there are no treatments available. I know better.

3. There are no approved drugs for Frontotemporal Dementia. That is simply because the disease is rare compared to other forms of dementia, and there is no economic incentive to develop or test a drug specifically for FTD. This does not mean that the drugs currently available for other approved uses are not effective in treating FTD, only that they have not been tested specifically for FTD. It does not mean new drugs will not be developed.  Which brings me to Herbal and Homeopathic Remedies.  No Herbal Remedy (and I use the term in its broadest sense) is approved by our health care system for treating anything - and that would include eating limes to cure Scurvy.  The way the law is right now in the United States if it said on a bag of limes at the grocery store that they cured Scurvy they would be classified as a drug by the FDA. Limes cure Scurvy! How is that for a dietary supplement treating a disease? To me saying there are no approved drugs is not the same as saying that there are no drugs available which would be effective for FTD.

4. Frontotemporal Dementia is progressive. What they are saying is that you get it, and it gets worse, and worse, and then eventually you die. Yuppers! To me that means if you sit around on your ass doing exactly what you were doing that got you into this mess it is progressive. I am sure that in that case it is true. Really now! Nobody can even agree on how to describe FTD, and they are out there telling us it is progressive like it is carved in stone. Maybe it is my demented logic, but I do not look at it that way at all. I do not believe that the progression is inevitable. It is not natural for my brain to be dying, and dying particularly fast in the frontal and temporal regions. Something has caused this to happen. It is unlikely it is genetic in my case, so that points to something behavioral or environmental. Well! Duh! If something behavioral or environmental can cause it, it can damn-well un-cause it too! If it can't be reversed, maybe it can be slowed down, or arrested. Nobody really knows because nobody has really looked yet.

So again -

So what does all this pessimistic crap mean to me with FTD?
What am I going to do?

For as long as I can, and as best that I can, I am going to search for things that help, and I have no intention of just sitting around on my ass waiting for somebody else to figure it out ten years after I am dead. Maybe it is futile. Maybe false hope. Maybe just something to occupy my time.  Maybe it will work. Maybe I will find something that slows the progress. Maybe I will find something that makes my day-to-day existence easier. Maybe I will find something that reverses some of the symptoms. Maybe I will find a cure. Maybe nothing will work, and I will march slowly towards that inevitable long night. In any case, as long as I can, I am going to fight! I am not yet ready to just give up and go quietly.

Do Not Go Gentle Into That Good Night

Do not go gentle into that good night,
Old age should burn and rave at close of day;
Rage, rage against the dying of the light.

Though wise men at their end know dark is right,
Because their words had forked no lightning they
Do not go gentle into that good night.

Good men, the last wave by, crying how bright
Their frail deeds might have danced in a green bay,
Rage, rage against the dying of the light.

Wild men who caught and sang the sun in flight,
And learn, too late, they grieved it on its way,
Do not go gentle into that good night.

Grave men, near death, who see with blinding sight
Blind eyes could blaze like meteors and be gay,
Rage, rage against the dying of the light.

And you, my father, there on that sad height,
Curse, bless, me now with your fierce tears, I pray.
Do not go gentle into that good night.
Rage, rage against the dying of the light.

Dylan Thomas

(Dear Rodney, Thank you. May you rest in peace. You will always have my respect. - Lee)

...fight like a fish in a blender ...but tomorrow, I think I will sleep in...

Comments are welcome.

Monday, December 6, 2010

Milestone - 5000 page views

Sometime around December 7, 2010 there were over 5000 page views on this blog since the time it was possible to gain such valuable information. I think they started tracking it around June, and my blog got listed on the search engines sometime in July. Anyway - It is nice to know that somebody out there visits now an then. I hope you find something here for you.

Here are a few of the places (in arbitrary but somewhat random order) where readers are interested in reading about bvFTD and found their way to my Frontotemporal Dementia bvFTD blog:
United States
United Kingdom

Below are a few of the search terms that people used in Google, Yahoo, or even that other one to find my blog.
dysexecutive syndrome symptoms
dementia and lexapro
bvftd stages
cider stopped bubbling
dementia ritalin
dysexecutive syndrome
frontotemporal dementia alternative treatments

Next to Google, the AFTD web site refers the most readers to my blog.

So, all I can say is,
Thank you! Over 5000 page visits! How cool is that?!

Read ...Comment... and don't forget to visit Amazon and buy a bunch of stuff ...but mostly, just ...Thanks

Some days are better than others.

Please visit again soon.